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Iron overload robustly induced hepatic tissue inflammation by upregulating IL-6, IL-1, and TNF- mRNA expression, while theaflavins supplementation remarkably attenuated iron overload induced hepatic tissue inflammation by downregulating IL-6, IL-1, and TNF- mRNA expression (Fig

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Randomized Clinical Trial of How Long-Term Glutathione Supplementation Offers Protection from Oxidative Damage and Improves HbA1c in Elderly Type 2 Diabetic Patients